AV Blocks Explained: First Degree, Mobitz Type One, Mobitz Type Two and Complete Heart Block Recognition

AV Blocks Explained: First Degree, Mobitz Type One, Mobitz Type Two and Complete Heart Block Recognition

🫀 ECG Foundations • Conduction Blocks

AV Blocks: First Degree, Mobitz Type One, Mobitz Type Two, Complete Heart Block

From a benign delay to a full conduction “divorce” — learn to recognise atrioventricular block patterns fast, understand what they mean physiologically, and know what matters first at the bedside.

Some rhythms do not race. They do not flutter. They do not fibrillate.

They simply pause — and that pause tells you where the message between atrium and ventricle is getting delayed… or lost.

Read AV block as “conduction physiology in motion”, not just ink on paper. 🫀
Editorial medical photo of an ECG monitor and clinical environment with negative space for title

What is an atrioventricular block?

An atrioventricular (AV) block is a conduction problem between atria and ventricles. The sinoatrial node may fire normally. The atria may depolarise normally. But somewhere between atrium and ventricle, the impulse is delayed or fails to conduct.

Unlike atrial fibrillation or atrial flutter, the issue is not chaos. It is interruption.


Normal conduction (the baseline story)

Normal ECG logic is simple:

  • Sinoatrial node fires
  • P wave appears (atrial depolarisation)
  • AV node delays conduction slightly (this is normal physiology)
  • QRS appears (ventricular depolarisation)

The PR interval normally ranges from 120 to 200 milliseconds. When the PR interval lengthens beyond that, or when impulses fail to conduct, AV block begins.


First-degree AV block

Delayed — but delivered.

ECG criteria

  • PR interval greater than 200 milliseconds
  • Every P wave conducts to a QRS
  • No dropped beats
  • Rhythm typically regular
🟢 What is happening?

Conduction is slowed (often within the atrioventricular node), but no impulse is lost. Think of a train that arrives late… but always arrives.

Clinical context

Often seen with increased vagal tone and with atrioventricular nodal blocking drugs (beta blockers, non-dihydropyridine calcium channel blockers, digoxin). It may also appear in inferior myocardial infarction. Most patients are asymptomatic.

Flowchart: recognising first-degree AV block

P wave present before every QRS?
      ↓ Yes
PR interval greater than 200 ms?
      ↓ Yes
No dropped beats?
      ↓ Yes
→ First-degree AV block

Mobitz Type One (Wenckebach)

Progressive fatigue.

ECG criteria

  • Progressive PR prolongation
  • Followed by a dropped QRS
  • Then the cycle repeats
🟡 What is happening?

The atrioventricular node conducts more and more slowly with each beat… until one impulse fails to conduct. Then it “resets”. It is organised, patterned, and usually atrioventricular nodal in origin.

Common settings

Young individuals with high vagal tone, sleep, inferior myocardial infarction, or medication effects. Many patients remain stable and asymptomatic.

Flowchart: recognising Mobitz Type One

PR interval changes beat to beat?
      ↓ Yes
Does PR progressively lengthen?
      ↓ Yes
Is there a dropped QRS after the progressive lengthening?
      ↓ Yes
→ Mobitz Type One (Wenckebach)

Mobitz Type Two

Sudden failure.

ECG criteria

  • PR interval is constant (for the conducted beats)
  • Dropped QRS complexes occur unexpectedly
  • No progressive PR prolongation
đź”´ Why Mobitz Type Two matters

Mobitz Type Two usually indicates disease below the atrioventricular node (His–Purkinje system). It can deteriorate into complete heart block. It is a pacing problem until proven otherwise.

Common causes

Anterior myocardial infarction, degenerative conduction disease, post-surgical injury, infiltrative disease. Symptoms may range from none to syncope depending on ventricular response and escape rhythm reliability.

Flowchart: recognising Mobitz Type Two

PR interval fixed?
      ↓ Yes
Sudden dropped QRS?
      ↓ Yes
No progressive lengthening beforehand?
      ↓ Yes
→ Mobitz Type Two

Complete heart block (Third-degree AV block)

Electrical divorce.

ECG criteria

  • P waves and QRS complexes are both present
  • No consistent relationship between P waves and QRS complexes
  • Atrioventricular dissociation with an escape rhythm
âš« What is happening?

The atria and ventricles beat independently. The atria follow the sinoatrial node. The ventricles rely on an escape rhythm — often slow and unreliable. This can produce hypotension, syncope, heart failure, and sudden cardiac arrest.

Flowchart: recognising complete heart block

Are P waves present?
      ↓ Yes
Are QRS complexes present?
      ↓ Yes
Is there any consistent relationship between P and QRS?
      ↓ No
→ Complete (Third-degree) AV block

Clinical approach to atrioventricular block

Step 1 — assess stability first

Prioritise physiology. Unstable features include hypotension, altered mental status, ischaemic chest pain, and acute heart failure. If unstable, proceed to urgent bradycardia management and pacing strategies as appropriate.

Step 2 — classify the pattern

Use one practical rule: what is the PR doing and are beats dropped?

  • First-degree: PR prolonged, no dropped beats
  • Mobitz Type One: PR progressively prolongs, then drops
  • Mobitz Type Two: PR fixed, sudden drops
  • Complete block: P waves and QRS are independent
đź§­ Localisation pearl

First-degree and Mobitz Type One are often atrioventricular nodal. Mobitz Type Two and many complete blocks are often infranodal (His–Purkinje). Infranodal disease is usually more dangerous.


Management overview (first-line concepts)

  • First-degree: usually observation; review drugs and reversible causes
  • Mobitz Type One: observation unless symptomatic; treat reversible triggers
  • Mobitz Type Two: treat as high risk; pacing is often required
  • Complete heart block: pacing is typically required urgently
The message is sent.
The message delays.
The message fails.

And the pulse tells you which chapter you are in.

Perioperative considerations 🏥

In theatre and critical care, atrioventricular block may appear due to increased vagal tone, myocardial ischaemia, electrolyte disturbance, drug effects, hypoxia, and procedural stimulation. In this setting, a “new block” is often a physiologic alarm — the conduction system is revealing stress.

đź§Ż Perioperative mindset

If a block appears during anaesthesia, ask: what is provoking vagal tone or ischaemia right now? Treat the trigger while you stabilise the rhythm.


Summary table

Block type PR interval pattern Dropped beats P–QRS relationship Risk level
First-degree Prolonged (greater than 200 milliseconds) No One to one conduction Usually low
Mobitz Type One Progressively prolonging Yes Patterned Often low
Mobitz Type Two Fixed (conducted beats) Yes Sudden failure High
Complete heart block Variable Independent rhythms No relationship (AV dissociation) Very high

References

  1. Kusumoto FM, et al. 2018 ACC/AHA/HRS Guideline on Bradycardia and Cardiac Conduction Delay. Circulation. https://www.ahajournals.org
  2. Surawicz B, Knilans TK. Chou’s Electrocardiography in Clinical Practice. (Textbook)
  3. Braunwald E. Braunwald’s Heart Disease: A Textbook of Cardiovascular Medicine. (Textbook)
  4. UpToDate. Atrioventricular block: Clinical manifestations and management. https://www.uptodate.com
  5. American Heart Association. Bradyarrhythmias and conduction blocks resources. https://www.heart.org
  6. European Society of Cardiology. Cardiac pacing and resynchronization therapy guidance. https://www.escardio.org
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